A case of pseudohypoparathyroidism presenting as epilepsy with increased parathyroid hormone-like activity in urine.

Secondary hyperparathyroidism in pseudohypoparathyroidism was suggested by Kolb (1962) based mainly on bone X-ray findings characteristic to parathyroid hyperfunction such as subperiosteal resorption and osteitis fibrosa. We have recently experienced a case of typical pseudohypoparathyroidism in which the parathyroid hormone-like calcium-mobilizing activity in urine was increased, although bone changes failed to show characteristic changes of parathyroid hormone excess.